<strong>My goals:</strong>
(1) Immediate term: to quickly, but gently cure my cat Wilhelm’s blocked common bile duct (acute condition) that is causing him pain and dangerously affecting his health.
(2) Longer term: to cure what has been a long-standing pattern of digestive upset—one that I suspect was a long-simmering precursor to the new acute condition.
<strong>The chief complaint: </strong>obstructed common bile duct (the duct that carries bile from the liver, through the gallbladder, and to the small intestine).
<strong>When did it start?</strong> First symptoms manifested at 3 am on Thursday, 6/28.
<strong>How often does it happen? </strong>This is a newly identified dis-ease for Wilhelm. However, the first set of symptoms he exhibited was recognizable to me, because I had seen him exhibit them and had treated them three times previously in the last two years. The second set of symptoms was brand new. (See below.)
<strong>What else was going on when it started? </strong>I had been having an extraordinarily stressful (emotionally stressful) days before his first symptom appeared.
<strong>What’s changed? What’s new? </strong>Relevant background: Since 2012, Wilhelm has frequently vomited hairballs and regurgitated undigested food. He is also frequently constipated, and he has food sensitivities.
A change to a homemade diet in 2015 has reduced the vomiting and regurgitation episodes where he vomits multiple hairballs and undigested food a few to many times over the course one to ten days. He frequently goes for months without vomiting or regurgitation at all.
He also still experiences constipation from time to time, and eating anything other than his normal diet will trigger severe itching in his ears and head.
My veterinarian says this collection of symptoms suggests he has irritable bowel syndrome (IBS) or irritable bowel disorder (IBD).
When he's his usual self, Wilhelm is a very easygoing, affectionate, cuddly, sweet cat who is very bonded with me. He is usually shy of new people, but warms up quickly. He can be vocal when he wants something. He likes to be warm, seeking sunbeams and heating vents and sleeping under the bedcovers with me in cold weather.
He loves to chew on plastic bags. He also will seek out and chew on cardboard when he is hungry or his stomach is upset. He is prone to vomiting more often in March and November, when he tends to shed more than usual.
Wilhelm is a white and gray domestic shorthair mix. I adopted him from an animal shelter in early July 2010. A stray, he was estimated to be four years old.
<em><strong>First set of symptoms for this new acute dis-ease:</strong> </em>
At 3 am, Wilhelm vomited up a small hairball and a lot of undigested food from the meal he ate around 10:30 pm the night before. After that he ate a bit of food, then regurgitated undigested food and plain bile five times before 6 am.
Between barfing episodes he was restless and pacing throughout the house. When he tried to sit or meatloaf, he did so in places that were not his usual sleeping/resting spots. He also sought out cardboard boxes to bite and gnaw on.
He was not vocalizing, which is atypical for him. He did not want to be touched—also atypical. He was not interested in the heating disc I warmed up and offered to him. He stayed on the floor and did not want to jump on the bed or chairs.
This is a symptom pattern he has previously exhibited on three different occasions in late 2016 and 2017. Each time, he was given Cerenia (maropitant citrate), which successfully stop the vomiting and allowed him to rest.
Recognizing this symptom pattern from previous occasions, at 6 am I gave him a 12 mg pill of Cerenia, which my vet had previously supplied. My goal was to stop the vomiting before he became dehydrated, which would mean needing to go to the vet for sub-cutaneous fluids.
I also gave him some energy therapy.
(Addressing the recurring pattern of vomiting/regurgitation episodes with homeopathy was something I intended to explore after taking the “Vitality, Balance and Homeopathy” course.)
<em><strong>Second set of symptoms for this new acute dis-ease:</strong></em>
Wilhelm’s vomiting stopped after he received the Cerenia tablet, and he was able to lie down and rest for about 45 minutes.
But then, around 7 am, he began visiting and revisiting the litter box—four times in 30 minutes. After one visit, I found a single small, hard poop nugget. After another, I found what looked like a thick, yellowish liquid; I couldn’t tell if it was atypical urine or very weirdly colored diarrhea.
He was also breathing through an open mouth, with his tongue sticking out, off and on. His tongue looked to be a darker, purplish hue instead of its usual pale pink.
<strong>Veterinary details:</strong>
Wilhelm’s physical exam suggested he had pain in his belly. He received a painkiller (buprenorphine, a narcotic) and an ultrasound.
The painkiller made him glassy-eyed, subdued, and very droolly.
I gave him energy therapy while we were waiting for the ultrasound vet. I could discern no physical response to the energy therapy, because Wilhelm was very subdued after receiving the painkiller. But I detected that a few of his major energy centers were compromised before the energy therapy, and open and balanced after the energy therapy.
The ultrasound revealed that his common bile duct (in the gallbladder, bringing bile from the liver to the small intestine) contained what the vet called “small, mineralized sludge.” That sludge was extending up into the corresponding liver duct. The bile duct was also enlarged to twice its size.
However, the ultrasound did not show any of the classic bowel signs of IBD.
The vet also ran blood work. Results:
RBC: 11.10 M/uL
HCT: 54.1% (high)
HGB: 17.7 g/dL (high)
MCV: 48.7 fL
MCH: 15.9 pg
MCHC: 32.7 g/dL
RDW: 26.4%
%RETIC: 0.1%
RETIC: 5.6 K/uL
WBC: 6.74 K/uL
%NEU: 60.9%
%LYM: 37.5%
%MONO: 0.7%
%EOS: 0.6%
%BASO: 0.3%
NEU: 4.10 K/uL
LYM: 2.53 K/uL
MONO: 0.05 K/uL
EOS: 0.04 K/uL (low)
BASO: 0.02 K/uL
PLT: 267 K/uL
MPV: 14.2 fL
PCT: 0.38%
GLU: 213 mg/dL (high)
CREA: 1.4 mg/dL
BUN: 22 mg/dL
BUN/CREA: 16
PHOS: 3.9 mg/dL
CA: 8.5 mg/dL
TP: 8.0 g/dL
ALB: 3.4 g/dL
GLOB: 4.6 g/dL
ALB/GLOG: 0.7
ALT: 2770 U/L (dangerously high)
ALKP: 122 U/L (high)
GGT: 0 U/L
TBIL: 1.6 mg/dL (high)
CHOL: 277 mg/dL (high)
AMYL: 1073 U/L
LIPA: 3069 U/L (high)
Na: 165 mmol/L
K: 3.5 mmol/L
Na/K: 47
Cl: 120 mmol/L
Osm Calc: 333 mmol/kg
The general vet said the next step would be to see a veterinary internist. The internist recommended to me could not see him until the next day (Friday, 6/29).
I chose to have Wilhelm spend last night (Thursday, 6/28) at a veterinary ER.
At the ER, he was given painkillers again and, using a feeding tube, they ensured that he got some food/nutrients. By doing this, they hoped that his metabolic system would naturally begin to start moving some of the built-up sediment out of the common bile duct. They also gave him subcutaneous fluids to maintain his hydration.
The internist’s first step will be to get an ultrasound focusing just on the gallbladder and liver, to see how much sediment there is in the bile duct today, after his treatment overnight. She will suggest next treatment steps based on the findings of that ultrasound.
(1) Immediate term: to quickly, but gently cure my cat Wilhelm’s blocked common bile duct (acute condition) that is causing him pain and dangerously affecting his health.
(2) Longer term: to cure what has been a long-standing pattern of digestive upset—one that I suspect was a long-simmering precursor to the new acute condition.
<strong>The chief complaint: </strong>obstructed common bile duct (the duct that carries bile from the liver, through the gallbladder, and to the small intestine).
<strong>When did it start?</strong> First symptoms manifested at 3 am on Thursday, 6/28.
<strong>How often does it happen? </strong>This is a newly identified dis-ease for Wilhelm. However, the first set of symptoms he exhibited was recognizable to me, because I had seen him exhibit them and had treated them three times previously in the last two years. The second set of symptoms was brand new. (See below.)
<strong>What else was going on when it started? </strong>I had been having an extraordinarily stressful (emotionally stressful) days before his first symptom appeared.
<strong>What’s changed? What’s new? </strong>Relevant background: Since 2012, Wilhelm has frequently vomited hairballs and regurgitated undigested food. He is also frequently constipated, and he has food sensitivities.
A change to a homemade diet in 2015 has reduced the vomiting and regurgitation episodes where he vomits multiple hairballs and undigested food a few to many times over the course one to ten days. He frequently goes for months without vomiting or regurgitation at all.
He also still experiences constipation from time to time, and eating anything other than his normal diet will trigger severe itching in his ears and head.
My veterinarian says this collection of symptoms suggests he has irritable bowel syndrome (IBS) or irritable bowel disorder (IBD).
When he's his usual self, Wilhelm is a very easygoing, affectionate, cuddly, sweet cat who is very bonded with me. He is usually shy of new people, but warms up quickly. He can be vocal when he wants something. He likes to be warm, seeking sunbeams and heating vents and sleeping under the bedcovers with me in cold weather.
He loves to chew on plastic bags. He also will seek out and chew on cardboard when he is hungry or his stomach is upset. He is prone to vomiting more often in March and November, when he tends to shed more than usual.
Wilhelm is a white and gray domestic shorthair mix. I adopted him from an animal shelter in early July 2010. A stray, he was estimated to be four years old.
<em><strong>First set of symptoms for this new acute dis-ease:</strong> </em>
At 3 am, Wilhelm vomited up a small hairball and a lot of undigested food from the meal he ate around 10:30 pm the night before. After that he ate a bit of food, then regurgitated undigested food and plain bile five times before 6 am.
Between barfing episodes he was restless and pacing throughout the house. When he tried to sit or meatloaf, he did so in places that were not his usual sleeping/resting spots. He also sought out cardboard boxes to bite and gnaw on.
He was not vocalizing, which is atypical for him. He did not want to be touched—also atypical. He was not interested in the heating disc I warmed up and offered to him. He stayed on the floor and did not want to jump on the bed or chairs.
This is a symptom pattern he has previously exhibited on three different occasions in late 2016 and 2017. Each time, he was given Cerenia (maropitant citrate), which successfully stop the vomiting and allowed him to rest.
Recognizing this symptom pattern from previous occasions, at 6 am I gave him a 12 mg pill of Cerenia, which my vet had previously supplied. My goal was to stop the vomiting before he became dehydrated, which would mean needing to go to the vet for sub-cutaneous fluids.
I also gave him some energy therapy.
(Addressing the recurring pattern of vomiting/regurgitation episodes with homeopathy was something I intended to explore after taking the “Vitality, Balance and Homeopathy” course.)
<em><strong>Second set of symptoms for this new acute dis-ease:</strong></em>
Wilhelm’s vomiting stopped after he received the Cerenia tablet, and he was able to lie down and rest for about 45 minutes.
But then, around 7 am, he began visiting and revisiting the litter box—four times in 30 minutes. After one visit, I found a single small, hard poop nugget. After another, I found what looked like a thick, yellowish liquid; I couldn’t tell if it was atypical urine or very weirdly colored diarrhea.
He was also breathing through an open mouth, with his tongue sticking out, off and on. His tongue looked to be a darker, purplish hue instead of its usual pale pink.
<strong>Veterinary details:</strong>
Wilhelm’s physical exam suggested he had pain in his belly. He received a painkiller (buprenorphine, a narcotic) and an ultrasound.
The painkiller made him glassy-eyed, subdued, and very droolly.
I gave him energy therapy while we were waiting for the ultrasound vet. I could discern no physical response to the energy therapy, because Wilhelm was very subdued after receiving the painkiller. But I detected that a few of his major energy centers were compromised before the energy therapy, and open and balanced after the energy therapy.
The ultrasound revealed that his common bile duct (in the gallbladder, bringing bile from the liver to the small intestine) contained what the vet called “small, mineralized sludge.” That sludge was extending up into the corresponding liver duct. The bile duct was also enlarged to twice its size.
However, the ultrasound did not show any of the classic bowel signs of IBD.
The vet also ran blood work. Results:
RBC: 11.10 M/uL
HCT: 54.1% (high)
HGB: 17.7 g/dL (high)
MCV: 48.7 fL
MCH: 15.9 pg
MCHC: 32.7 g/dL
RDW: 26.4%
%RETIC: 0.1%
RETIC: 5.6 K/uL
WBC: 6.74 K/uL
%NEU: 60.9%
%LYM: 37.5%
%MONO: 0.7%
%EOS: 0.6%
%BASO: 0.3%
NEU: 4.10 K/uL
LYM: 2.53 K/uL
MONO: 0.05 K/uL
EOS: 0.04 K/uL (low)
BASO: 0.02 K/uL
PLT: 267 K/uL
MPV: 14.2 fL
PCT: 0.38%
GLU: 213 mg/dL (high)
CREA: 1.4 mg/dL
BUN: 22 mg/dL
BUN/CREA: 16
PHOS: 3.9 mg/dL
CA: 8.5 mg/dL
TP: 8.0 g/dL
ALB: 3.4 g/dL
GLOB: 4.6 g/dL
ALB/GLOG: 0.7
ALT: 2770 U/L (dangerously high)
ALKP: 122 U/L (high)
GGT: 0 U/L
TBIL: 1.6 mg/dL (high)
CHOL: 277 mg/dL (high)
AMYL: 1073 U/L
LIPA: 3069 U/L (high)
Na: 165 mmol/L
K: 3.5 mmol/L
Na/K: 47
Cl: 120 mmol/L
Osm Calc: 333 mmol/kg
The general vet said the next step would be to see a veterinary internist. The internist recommended to me could not see him until the next day (Friday, 6/29).
I chose to have Wilhelm spend last night (Thursday, 6/28) at a veterinary ER.
At the ER, he was given painkillers again and, using a feeding tube, they ensured that he got some food/nutrients. By doing this, they hoped that his metabolic system would naturally begin to start moving some of the built-up sediment out of the common bile duct. They also gave him subcutaneous fluids to maintain his hydration.
The internist’s first step will be to get an ultrasound focusing just on the gallbladder and liver, to see how much sediment there is in the bile duct today, after his treatment overnight. She will suggest next treatment steps based on the findings of that ultrasound.